Calcium build-up in cells results in diverse range of changes in the brain. There are two significant changes that develop conditions for Alzheimer’s disease.

Specific increases in the amyloid precursor protein, BACE1 and amyloid beta protein aggregates Less specific neurodegenerative changes including hyperphosphorylated tau protein and neurofibrillary tangles, cell death produced by apoptosis or by destructive autophagy, synaptic dysfunction produced by changes in neuronal structures required for synapsis between brain neurons, oxidative stress and inflammation

Pulsed electronically generated electromagnetic fields (EMFs) used for wireless communication are coherent producing strong electric and magnetic forces that act in the cells of our bodies primarily via activation of voltage-gated calcium channels (VGCCs). VGCC activation produces rapid increases in intracellular calcium levels. Therefore, EMF exposures produce changes with lead to excessive intracellular calcium. This buildup explains the effects on the brain in Alzheimer’s disease.

These EMF induced changes to intracellular calcium levels have been demonstrated in animal models of Alzheimer’s disease. Research has shown the involvement of two pathways that lead to Alzheimer’s disease. Each of the two pathways producing pathophysiological effects following EMF exposure are important in Alzheimer’s causation: the excessive calcium signaling pathway and the peroxynitrite/oxidative stress/inflammation pathway.

Professor Martin L. Pall, at the Washington State University has been studying this phenomenon for a decade. “EMFs act via peak electric and time varying magnetic forces at a nanosecond time scale.”, says Professor Pall. Such peaks are vastly increased with each increase in pulse modulation produced by smarter cell phones, smart meters, smart cities and radar in self driving vehicles. “Any of these may produce the ultimate nightmare – extremely early onset Alzheimer’s Disease.”

These findings provide powerful evidence that EMFs can cause Alzheimer’s disease. Human and animal studies add still more evidence to the calcium hypothesis of Alzheimer’s disease

Human genetic and pharmacological studies show that elevated VGCC activity causes increased Alzheimer’s incidence. Consequently, it is not just calcium that is important. VGCC activity, which is directly greatly increased by EMF exposure, is build-up also important in causing Alzheimer’s.

12 recent occupational exposure assessments have found that people having occupational EMF exposures have higher Alzheimer’s incidence. Some studies suggest that EMFs shorten the normal 25-year latency period of Alzheimer’s disease.

Source: This news is originally published by medical

By Web Team

Technology Times Web team handles all matters relevant to website posting and management.